(1) anemia is uremic patients must have symptoms. The degree of anemia and uremia (kidney function) degree parallel, erythropoietin (epo) reduction as the main reason.
(2) bleeding tendency may be manifested as skin, mucosal bleeding, and increased platelet destruction, prolonged bleeding, etc., which may be caused by toxins, dialysis correctable.
(3) abnormal white blood leukopenia, chemotaxis, phagocytosis and bactericidal capacity weakened, prone to infection, post-dialysis can improve.
Oliguric renal potassium excretion is reduced. Organism increased catabolism, metabolic acidosis, transfer to the extracellular K +, using retention potassium-sparing diuretics or angiotensin converting enzyme inhibitors, can cause severe hyperkalemia. Manifested as lethargy, severe arrhythmias, or even cardiac arrest. If you eat less, potassium intake, nausea. Vomiting, diarrhea and long-term application of potassium sparing diuretics. Prone to hypokalemia. As fatigue, muscle weakness, abdominal distension, limb paralysis. In severe cases of severe arrhythmias and respiratory muscle paralysis.
3. hyponatremia, chronic renal failure patients on sodium regulation is poor.
Because renal tubular sodium absorption dysfunction, coupled with some other factors, such as uniforms diuretics, diarrhea, long-term consumption of salt-free diet. Prone to hyponatremia. Due to the loss of sodium and water, causing hypovolemia. Loss of sodium leads to rapid deterioration of renal function. Therefore hyponatremia often can a patient had stable disease uremic symptoms appear. Patients often feel fatigue, weakness, dizziness, orthostatic hypotension, muscle twitching, thready pulse and speed, severe shock may occur. Conversely, if the sodium intake, the body will be retention, causing edema, hypertension, severe heart failure can occur.
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