When renal failure, glomerular filtration rate decreased ability impaired renal excretion of metabolites, protein catabolism of AGEs accumulate in the blood, and the composition of uremic toxins main component of blood urea nitrogen, creatinine, guanidine, polyamines and some of the molecules.
Low-protein diet can reduce the generation and accumulation of protein metabolism, thereby reducing the residual renal units high metabolic state, improve glomerular hemodynamic abnormalities, reduce glomerular hyperfiltration state, delaying glomerulosclerosis and renal progress dysfunction. Therefore, low-protein diet therapy in patients with chronic renal insufficiency primary means of non-dialysis treatment.
It should be noted that long-term low-protein diet can cause renal failure in patients with malnutrition, affecting the quality of life, in order to avoid this situation, you can take high-quality low-protein diet plus service compound α- keto acid (open with) diet regimen because the amino acid compound α- keto acid precursor, via transamination in vivo conversion to the corresponding essential amino acids, provide raw materials for the protein synthesis, and does not produce metabolites urea nitrogen, creatinine, etc. In addition, since the necessary complement amino acids, so that the body can take full advantage of the imbalance and excess non-essential amino acids to synthesize proteins, correct deficiencies body protein. Taking the same open and with a low-protein diet can reduce high glomerular filtration to protect renal units, so as to achieve slowing the deterioration of chronic renal failure purpose.
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