Benign arteriolar nephrosclerosis hypertension essential hypertension-induced
nephropathy system (also known as hypertension, renal arteriosclerosis) and
malignant small renal arteries harden and accompanied by corresponding clinical
manifestations of the disease, hypertension and renal failure.
More than a perennial history of hypertension, renal tubular damage much
earlier than the glomeruli, nocturia, urine concentration dysfunction, urinary
changing light, mild proteinuria, may have microscopic hematuria and casts,
often Other target organ complications of hypertension.
Hypertensive nephropathy examination findings: that the general sustained
increase in blood pressure (/ Kpa / mmHg above); some of the eyelids and / or
lower extremity edema, heart community to expand the like; most arteriosclerotic
retinopathy, when the fundus striped flame-shaped hemorrhage The soft exudates
and cotton wool, support and malignant renal atherosclerosis, hypertensive
encephalopathy diagnosis with the nervous system may have a corresponding
locating signs.
In clinical practice, according to the patient's condition and laboratory
tests, usually divided into the following several of hypertensive
nephropathy:
Ⅰ period - microalbuminuria: rate of urinary albumin excretion characterized
by abnormal. Normal renal function, urine protein negative;
Ⅱ - A clinical proteinuria: A urine protein positive, 24h urinary protein
excretion "0.5g characterized with normal renal function.
Ⅲ stage - renal insufficiency: taking Ccr decreased, SCr elevated features. 1
Non-dialysis and dialysis (uremia).
Non-dialysis: Ccr at 40 ~ 10ml / min, 133μmol / L 707μmol / L.
Dialysis (uremia): Ccr <10ml / min. Scr> 707μmol / L.
If you have any questions you want advice, you can contact us by email:
chinakidneyhospital@gmail.com or Tel: +8613633219293.
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