1. The history of acute pyelonephritis history as reference of diagnosis, but
cannot serve as the basis.For the majority of patients with non obstructive
chronic pyelonephritis, can always without history of urinary tract infection,
but also do not have other history of kidney disease.Have disease often
obscurity, nitrogen qualitative hematic disease symptoms for starting symptoms,
diagnosis should be pay attention.
2. The clinical manifestations of urinary irritation symptoms of intermittent
reaction, generally lighter, less obvious acute pyelonephritis, often
accompanied by fatigue, loss of appetite, waist ache, can have low thermal or no
fever.Late can appear because of renal impairment of uremia symptom such as
dizziness, headache, nausea and vomiting.May also appear polyuria, nocturia
increased, low potassium, low serum sodium or chronic renal tubular
acidosis.Part of the insidious or atypical symptoms in patients, appropriate
attention.
3. The auxiliary examination
(1) routine urine, urine protein is commonly trace or a few.If the urine
protein > 3.0/24 hour, the tip of the disease.Urinary sediment can have a
small amount of red blood cells and white blood cells.If discover the white tube
type is helpful to diagnosis, but not unique to this disease.
(2)Urine culture: with acute pyelonephritis, but positive rate is low,
sometimes need to repeatedly check will obtain a positive result.Patients with
negatie urine bacteria culture type strains about 20% can be found in the virgin
pulp, the department of pathogenic bacteria in the function of antibacterial
drugs, and antibody, in order to adapt to bad environment and survive a variable
capacity, membrane rupture, but virgin pulp quality is still, once the
environment favorable to breeding.Bladder after sterilization antibody check
parcel bacteria positive urine culture and urine, help to diagnosis of the
disease, can be identified with cystitis.
(3) check: renal function usually has decreased renal tubular function (urine
concentration is reduced, phenol red excretion rate reduce, etc.), can be
increased urine, urinary potassium sodium excretion, metabolic acidosis;Periods
of urinary potassium can be increased.Late in glomerular function obstacle,
blood urea nitrogen and creatinine increased, leading to uremia.
(4) X-ray imaging: visible renal pelvis, calyces deformation bright shadow
irregular even smaller.
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